Sergio E. Baranzini, Pouya Khankhanian, Nikolaos A. Patsopoulos, Michael Li, Jim Stankovich, Chris Cotsapas, Helle Bach Søndergaard, Maria Ban, Nadia Barizzone, Laura Bergamaschi, David R. Booth, Dorothea Buck, Paola Cavalla, Elisabeth Gulowsen Celius, Manuel Comabella, Gıancarlo Comı, Alastair Compston, Isabelle Cournu‐Rebeix, Sandra D’Alfonso, Vincent Damotte, Lennox Din, Bénédicte Dubois, Irina Elovaara, Federica Esposito, Bertrand Fontaine, André Franke, An Goris, Pierre‐Antoine Gourraud, Christiane Graetz, Franca Rosa Guerini, Léna Guillot‐Noël, D Hafler, Hákon Hákonarson, Per Hall, Anders Hamsten, Hanne F. Harbo, Bernhard Hemmer, Jan Hillert, Anu Kemppinen, Ingrid Kockum, Keijo Koivisto, Malin Larsson, Mark Lathrop, Maurizio Leone, Christina M. Lill, Fabìo Macciardi, Roland Martinꝉ, Vittorio Martinelli, Filippo Martinelli Boneschi, Jacob L. McCauley, Kjell‐Morten Myhr, Paola Naldi, Tomas Olsson, Annette Oturai, Margaret A. Pericak‐Vance, Franco Perla, Mauri Reunanen, Janna Saarela, Safa Saker-Delye, Marco Salvetti, Finn Sellebjerg, Per Soelberg Sørensen, Anne Spurkland, Graeme J. Stewart, Bruce Taylor, Pentti J. Tienari, Juliane Winkelmann, Frauke Zipp, Adrian J. Ivinson, Jonathan L. Haines, Stephen Sawcer, Philip L. DeJager, Stephen L. Hauser, Jorge R. Oksenberg,
Multiple sclerosis (MS) is an inflammatory CNS disease with a substantial genetic component, originally mapped to only the human leukocyte antigen (HLA) region. In the last 5 years, a total of seven genome-wide association studies and one meta-analysis successfully identified 57 non-HLA susceptibility loci. Here, we merged nominal statistical evidence of association and physical evidence of interaction to conduct a protein-interaction-network-based pathway analysis (PINBPA) on two large genetic MS ...
Tópico(s): Genetic Associations and Epidemiology
2013 - Elsevier BV | The American Journal of Human Genetics