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The β‐Amyloid Peptide, Peptide Self‐Assembly, and the Emergence of Biological Activities

1997; Wiley; Volume: 814; Issue: 1 Linguagem: Inglês

10.1111/j.1749-6632.1997.tb46140.x

1749-6632

Carl W. Cotman,

Prion Diseases and Protein Misfolding

Annals of the New York Academy of SciencesVolume 814, Issue 1 p. 1-16 The β-Amyloid Peptide, Peptide Self-Assembly, and the Emergence of Biological Activities A New Principle in Peptide Function and the Induction of Neuropathology CARL W. COTMAN, CARL W. COTMAN Institute for Brain Aging and Dementia University of California, Irvine Irvine, California 92697Search for more papers by this author CARL W. COTMAN, CARL W. COTMAN Institute for Brain Aging and Dementia University of California, Irvine Irvine, California 92697Search for more papers by this author First published: 17 December 2006 https://doi.org/10.1111/j.1749-6632.1997.tb46140.xCitations: 16Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL REFERENCES 1 Kelly, J. W. 1996. Alternative conformations of amyloidogenic proteins govern their behavior. Curr. Opin. Struct. Biol. 6: 11– 17. 2 Cummings, B. J. & C. W. Cotman. 1995. Image analysis of beta-amyloid load in Alzheimer's disease and relation to dementia severity. 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