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Diaphyseal medullary stenosis with malignant fibrous histiocytoma: Further evidence for loss of heterozygosity involving 9p21‐22 in tumor tissue

2001; Wiley; Volume: 33; Issue: 3 Linguagem: Inglês

10.1002/gcc.10023

1098-2264

Koji Muroya, Gen Nishimura, Hideo Douya, Tadashi Hasegawa, Tsutomu Ogata,

Histiocytic Disorders and Treatments

Genes, Chromosomes and CancerVolume 33, Issue 3 p. 326-328 Letters to the Editor Diaphyseal medullary stenosis with malignant fibrous histiocytoma: Further evidence for loss of heterozygosity involving 9p21-22 in tumor tissue Koji Muroya, Koji Muroya Department of Pediatrics, Keio University School of Medicine, Tokyo, JapanSearch for more papers by this authorGen Nishimura, Gen Nishimura Department of Radiology, Nasu Chuo Hospital, Ohtawara, JapanSearch for more papers by this authorHideo Douya, Hideo Douya Division of Orthopedic Surgery, National Cancer Center Hospital, Tokyo, JapanSearch for more papers by this authorTadashi Hasegawa, Tadashi Hasegawa Division of Pathology, National Cancer Center Research Institute, Tokyo, JapanSearch for more papers by this authorTsutomu Ogata, Tsutomu Ogata Department of Pediatrics, Tokyo Electric Power Company Hospital, Tokyo, JapanSearch for more papers by this author Koji Muroya, Koji Muroya Department of Pediatrics, Keio University School of Medicine, Tokyo, JapanSearch for more papers by this authorGen Nishimura, Gen Nishimura Department of Radiology, Nasu Chuo Hospital, Ohtawara, JapanSearch for more papers by this authorHideo Douya, Hideo Douya Division of Orthopedic Surgery, National Cancer Center Hospital, Tokyo, JapanSearch for more papers by this authorTadashi Hasegawa, Tadashi Hasegawa Division of Pathology, National Cancer Center Research Institute, Tokyo, JapanSearch for more papers by this authorTsutomu Ogata, Tsutomu Ogata Department of Pediatrics, Tokyo Electric Power Company Hospital, Tokyo, JapanSearch for more papers by this author First published: 12 December 2001 https://doi.org/10.1002/gcc.10023Citations: 5Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onEmailFacebookTwitterLinkedInRedditWechat No abstract is available for this article. REFERENCES Hahn M, Matzen SE, Serth J, Pingoud A. 1995. Semiautomated quantitative detection of loss of heterozygosity in the tumor suppressor gene p53. Biotechniques 18: 1040–1047. CASPubMedWeb of Science®Google Scholar Hardcastle P, Nade S, Arnold W. 1986. Hereditary bone dysplasia with malignant change: Report of three families. J Bone Joint Surg Am 68: 1079–1089. Google Scholar Martignetti JA, Desnick RJ, Aliprandis E, Norton KI, Hardcastle P, Nade S, Gelb BD. 1999. Diaphyseal medullary stenosis with malignant fibrous histiocytoma: A hereditary bone dysplasia/cancer syndrome maps to 9p21-22. Am J Hum Genet 64: 801–807. 10.1086/302297 CASPubMedWeb of Science®Google Scholar Martignetti JA, Gelb BD, Pierce H, Picci P, Desnick RJ. 2000. Malignant fibrous histiocytoma: Inherited and sporadic forms have loss of heterozygosity at chromosome bands 9p21-22. Evidence for a common genetic defect. Genes Chromosomes Cancer 27: 191–195. 10.1002/(SICI)1098-2264(200002)27:2 3.0.CO;2-L CASPubMedWeb of Science®Google Scholar Simons A, Schepens M, Jeuken J, Sprenger S, van de Zande G, Bjerkehagen B, Forus A, Weibolt V, Molenaar I, van den Berg E, Myklebost O, Bridge J, van Kessel AG, Suijkerbuijk R. 2000. Frequent loss of 9p21 (p16(INK4A)) and other genomic imbalances in human malignant fibrous histiocytoma. Cancer Genet Cytogenet 118: 89–98. 10.1016/S0165-4608(99)00178-8 CASPubMedWeb of Science®Google Scholar Citing Literature Volume33, Issue3March 2002Pages 326-328 ReferencesRelatedInformation