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Idiopathic Intracranial Hypertension and Facial Palsy: Case Report and Review of the Literature

2010; SAGE Publishing; Volume: 25; Issue: 12 Linguagem: Inglês

10.1177/0883073810375849

1708-8283

Meropi Tzoufi, Alexandros Makis, Vasilios Grammeniatis, Iliada Nakou, Georgios Exarchakos, Ioannis Asproudis, Anastasia Zikou, Maria I. Argyropoulou, Antigone Siamopoulou-Mavridou,

Neurosurgical Procedures and Complications

We present the case of an 11-year-old obese girl who presented with idiopathic intracranial hypertension affecting first the lateral abducens nerve. She received acetazolamide, but 5 days later she developed lateral, peripheral facial palsy. Imaging evaluation was normal, which primarily excluded cerebral venous thrombosis and sustained the initial diagnosis. Despite some complicating factors (obesity, elevated intracranial pressure), prednisolone was administered for a short-term period to counteract the facial palsy. Ophthalmological residuals resolved within almost 1.5 months, while facial palsy receded after 4 months. Peripheral facial palsy is an extremely rare, but not unknown condition in idiopathic intracranial hypertension. As a symptom, it should be investigated thoroughly, primarily to exclude cerebral venous sinus thrombosis, before it can be attributed to idiopathic intracranial hypertension. As far as treatment is concerned, corticosteroids can be added to the initial treatment with acetazolamide, without worsening already elevated intracranial hypertension or ophthalmologic findings.