Artigo Acesso aberto Revisado por pares

Elevated middle cerebral artery peak systolic velocity without fetal anemia in a case of homozygous α–thalassemia‐1

2008; Wiley; Volume: 28; Issue: 1 Linguagem: Inglês

10.1002/pd.1914

ISSN

1097-0223

Autores

Karla Maguire, Anthony E. Johnson, Ching‐Nan Ou, Regina L. Lantin, Kenneth J. Moise,

Tópico(s)

Fetal and Pediatric Neurological Disorders

Resumo

Prenatal DiagnosisVolume 28, Issue 1 p. 72-74 Research LetterFree Access Elevated middle cerebral artery peak systolic velocity without fetal anemia in a case of homozygous α–thalassemia-1 Karla Maguire, Karla Maguire Department of Obstetrics and Gynecology, Division of Maternal-Fetal Medicine, Baylor College of Medicine, Houston, Texas, USASearch for more papers by this authorAnthony Johnson, Anthony Johnson Department of Obstetrics and Gynecology, Division of Maternal-Fetal Medicine, Baylor College of Medicine, Houston, Texas, USA Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USASearch for more papers by this authorChing-Nan Ou, Ching-Nan Ou Department of Pathology, Baylor College of Medicine, Houston, Texas, USASearch for more papers by this authorRegina L. Lantin, Regina L. Lantin Department of Pediatrics, Division of Pediatric Cardiology, Baylor College of Medicine, Houston, Texas, USASearch for more papers by this authorKenneth J. Moise, Corresponding Author Kenneth J. Moise [email protected] Department of Obstetrics and Gynecology, Division of Maternal-Fetal Medicine, Baylor College of Medicine, Houston, Texas, USA Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USADepartment of Obstetrics and Gynecology, Division of Maternal-Fetal Medicine, Baylor College of Medicine, 6620 Main Street, Suite 1100, Houston, Texas 77030, USA.Search for more papers by this author Karla Maguire, Karla Maguire Department of Obstetrics and Gynecology, Division of Maternal-Fetal Medicine, Baylor College of Medicine, Houston, Texas, USASearch for more papers by this authorAnthony Johnson, Anthony Johnson Department of Obstetrics and Gynecology, Division of Maternal-Fetal Medicine, Baylor College of Medicine, Houston, Texas, USA Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USASearch for more papers by this authorChing-Nan Ou, Ching-Nan Ou Department of Pathology, Baylor College of Medicine, Houston, Texas, USASearch for more papers by this authorRegina L. Lantin, Regina L. Lantin Department of Pediatrics, Division of Pediatric Cardiology, Baylor College of Medicine, Houston, Texas, USASearch for more papers by this authorKenneth J. Moise, Corresponding Author Kenneth J. Moise [email protected] Department of Obstetrics and Gynecology, Division of Maternal-Fetal Medicine, Baylor College of Medicine, Houston, Texas, USA Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USADepartment of Obstetrics and Gynecology, Division of Maternal-Fetal Medicine, Baylor College of Medicine, 6620 Main Street, Suite 1100, Houston, Texas 77030, USA.Search for more papers by this author First published: 10 January 2008 https://doi.org/10.1002/pd.1914Citations: 7AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onEmailFacebookTwitterLinkedInRedditWechat No abstract is available for this article. REFERENCES Chiu DHK. 2005. α-Thalassemia: Hb H disease and Hb Barts hydrops fetalis. Ann N Y Acad Sci 1054: 25–32. Dame C, Albers N, Hasan C, et al. 1999. Homozygous α-thalassaemia and hypospadias–common aetiology or incidental association? Long term survival of Hb Bart's hydrops syndrome leads to new aspects of counseling of α-thalassaemic traits. Eur J Pediatr 158: 217–220. Horton BF, Thompson RB, Dozy AM, et al. 1962. Inhomogeneity of hemoglobin. VI. The minor hemoglobin components of cord blood. Blood 20: 302–313. Imbar T, Lev-Sagie A, Cohen S, et al. 2006. Diagnosis, surveillance, and treatment of the anemic fetus using middle cerebral artery peak systolic velocity measurement. Prenat Diagn 26: 45–51. Lam YH, Tan MHY. 2002. Middle cerebral artery Doppler study in fetuses with homozygous α-thalassaemia-1 at 12–13 weeks gestation. Prenat Diagn 22: 56–58. Leung WC, Oepkes D, Seaward G, Ryan G. 2002. Serial sonographic findings of four fetuses with homozygous alpha-thalassemia-1 from 21 weeks onwards. Ultrasound Obstet Gynecol 19: 56–59. Mari G, Collaborative Group for Doppler Assessment of the Blood Velocity in Anemic Fetuses. 2000. Noninvasive diagnosis by Doppler ultrasonography of fetal anemia due to maternal red-cell alloimmunization. N Engl J Med 342: 9–14. Maberry MC, Klein VR, Boehm C, et al. 1990. Alpha-thalassemia: prenatal diagnosis and neonatal implications. Am J Perinatol 7: 356–358. Picklesimer AH, Oepkes D, Moise KJ Jr, et al. 2007. Determinants of the middle cerebral artery peak systolic velocity in the human fetus. Am J Obstet Gynecol 197(5): 526.e1–4. Randhawa ZI, Jones RT, Lie-Injo LE. 1984. Human hemoglobin Portland II. J Biol Chem 259: 7325–7330. Citing Literature Volume28, Issue1January 2008Pages 72-74 ReferencesRelatedInformation

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