Artigo Revisado por pares

Primdone, phenobarbital, and PEMA

1983; Lippincott Williams & Wilkins; Volume: 33; Issue: 3 Linguagem: Inglês

10.1212/wnl.33.3.283

ISSN

1526-632X

Autores

Blaise F. D. Bourgeois, W. E. Dodson, James A. Ferrendelli,

Tópico(s)

Pain Mechanisms and Treatments

Resumo

Neurotoxicity and protection against maximal electroshock and Metrazol seizures from primidone (PRM), phenobarbital (PB), and phenylethylmalonamide (PEMA) were determined in mice for each drug separately and expressed in terms of brain concentrations. Compared with PB, PEMA was 16 times less potent against electroshock and Metrazol seizures but only 8 times less toxic. Primidone was markedly less neurotoxic than PB and equally potent against electroshock, but PRM had no effect against Metrazol or bicuculline. PRM is a relatively nontoxic anticonvulsant with a different action than PB, and PEMA is both a weak and a relatively toxic anticonvulsant.

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