Revisão Revisado por pares

Dyslexia, Congenital Anomalies, and Immune Disorders: The Role of the Fetal Environmenta

1985; Wiley; Volume: 457; Issue: 1 Linguagem: Inglês

10.1111/j.1749-6632.1985.tb20796.x

ISSN

1749-6632

Autores

Peter O. Behan, Norman Geschwind,

Tópico(s)

Genetics and Neurodevelopmental Disorders

Resumo

Annals of the New York Academy of SciencesVolume 457, Issue 1 p. 13-18 Dyslexia, Congenital Anomalies, and Immune Disorders: The Role of the Fetal Environmenta PETER BEHAN, PETER BEHAN Department of Neurology Institute of Neurological Sciences University of Glasgow Glasgow G51 4TF, ScotlandSearch for more papers by this authorNORMAN GESCHWIND, NORMAN GESCHWIND Neurological Unit Beth Israel Hospital Boston, Massachusetts 02215Search for more papers by this author PETER BEHAN, PETER BEHAN Department of Neurology Institute of Neurological Sciences University of Glasgow Glasgow G51 4TF, ScotlandSearch for more papers by this authorNORMAN GESCHWIND, NORMAN GESCHWIND Neurological Unit Beth Israel Hospital Boston, Massachusetts 02215Search for more papers by this author First published: December 1985 https://doi.org/10.1111/j.1749-6632.1985.tb20796.xCitations: 31 a This work was supported in part by Grants 06209 and NS 17018 from the National Institute of Neurological and Communication Disorders and Stroke of the National Institutes of Health and by the Orton Research Fund and the Essel Foundation. b Deceased November 4, 1984 AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onEmailFacebookTwitterLinkedInRedditWechat References 1 Behan, W. M. H. & P. O. Behan 1980. A comparison between human polymyositis and the putative animal model, experimental allergic myositis. In Animal Models of Neurological Disease. F. C. Rose & P. O. Behan, Eds.: 72–89. Pitman. London . 2 Behan, W. M. H. & P. O. Behan 1984. Disturbance of regulatory lymphocyte subsets in polymyositis. Ann. Neurol. 15: 112–114. 3 Committee on Safety of Medicines. January 1983. Sodium valproate (Epilim) and congenital abnormalities. Current Problems No. 9.: 1–2. HMSO. London . 4 Drake, W. E. 1968. Clinical and pathological findings in a child with development learning disability, J. Learn. Disabil. 1: 9–25. 5 Feingold, J., N. Feingold & E. Bois 1980. Spina bifida and anencephaly. Geographic correlation with the HLA system. Tissue Antigens 15: 318–324. 6 Galaburda, A. M. 1983. Developmental dyslexia: Current anatomical research. Ann. Dyslexia 33: 41–58. 7 Galaburda, A. M. & T. L. Kemper 1979. Cytoarchitectonic abnormalities in developmental dyslexia: A case study. Ann. Neurol. 6: 94–100. 8 Geschwind, N. 1984. Immunological associations of Cerebral dominance. In Neuroimmunology. P. Behan & F. Spreafico, Eds.: 451–461. Raven Press. New York , NY . 9 Geschwind, N. & P. O. Behan 1982. Left-handedness: Association with immune disease, migraine and developmental learning disorders. Proc. Natl. Acad. Sci. USA 79: 5097–5100. 10 Geschwind, N. & P. O. Behan 1984. Laterality, hormones and immunity. In Cerebral Dominance: the Biological Foundations. N. Geschwind & A. Galaburda, Eds.: 211–224. Harvard University Press. Cambridge , MA . 11 Grove, D. I., S. J. O'Callaghan & T. O. Burston, & I. J. Forbes 1973. Immunological function in dystrophia myotonica. Br. Med. J. 3: 81–83. 12 Kolata, G. 1983. Math genius may have hormonal basis. Science 222: 1312. 13 Maddison, P., H. Mogavero, T. T. Provost & M. Reichlin 1979. 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Immunological diseases in pregnancy. In Progress in Allergy. P. Kallas et al. Eds. 23: 321–366. 21 Scott, J. S., P. J. Maddison, P. V. Raylor, E. Esscher, O. Scott & R. P. Skinner 1983. Connective-tissue disease, antibodies to ribonucleoprotein, and congenital heart block. New Engl. J. Med. 309: 209–212. 22 Tan, E. M. 1982. Autoantibodies to nuclear antigens (ANA): Their immunobiology and medicine. Adv. Immunol. 33: 167–240. 23 Thompson, A. M. P. 1968. Dystrophia cordis myotonica studied by serial histology of the pacemaker and conducting tissue. J. Pathol. Bacteriol. 96: 285–295. 24 Vannier, J. P., B. Cavelier, J. P. Martin, J. Lefort, L. Rivat & J. Feingold 1980. HLA, Pi, G, and Km phenotypes in a spina bifida population with myelomeningocele. Tissue Antigens 15: 501–504. 25 Veith, G. & H-K. Ziegler 1965. Fehlbildungen des Gehirnes bei Endokardofibroelastose. Beitr. Pathol. Anat. 132: 160–187. 26 Venables, P. J. W., P. R. Smith & R. N. Maini 1983. Purification and characterization of the Sjögren's syndrome A and B antigens. Clin. Exp. Immunol. 54: 731–738. 27 Wolin, S. L. & J. A. Steitz 1984. The Ro small cytoplasmic ribonucleoproteins: Indentification of the antigenic protein and its binding site on the Ro RNAs. Proc. Natl. Acad. Sci. USA. 81: 1996–2000. 28 Wood, L. O. 1983. Presentation at the American Thyroid Association, New Orleans , 1983. Citing Literature Volume457, Issue1Hope for a New NeurologyDecember 1985Pages 13-18 ReferencesRelatedInformation

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