Artigo Revisado por pares

A novel small deletion in PMP22 causes a mild hereditary neuropathy with liability to pressure palsies phenotype

2011; Wiley; Volume: 45; Issue: 1 Linguagem: Inglês

10.1002/mus.22201

ISSN

1097-4598

Autores

Carlos Casasnovas, Isabel Banchs, Laura Jorge, Maria Alberti, Yolanda Martínez–Campo, Mònica Povedano, Jordi Montero, Vı́ctor Volpini,

Tópico(s)

Neurological diseases and metabolism

Resumo

Abstract Introduction: In this study we examined a family with electrophysiological findings of hereditary neuropathy with liability to pressure palsies (HNPP) and a mild clinical presentation. Methods: Four members of a family were referred for diagnosis of HNPP. Electrophysiological studies included motor and sensory nerve conduction studies in the upper and lower extremities. Investigations of microsatellites, using polymorphic repeat markers flanking the gene, and multiplex ligation‐dependent probe amplification (MLPA) were performed for molecular studies. Results: The initial study of microsatellites did not detect any change, but MLPA demonstrated a small deletion of exon 5 in the PMP22 gene. Conclusion: Our findings demonstrate the important role of small deletions in the PMP22 gene in the etiology of HNPP with a normal microsatellite study. Muscle Nerve 45: 135–138, 2012

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