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Open surgical repair of tetralogy of Fallot in dogs

2001; American Veterinary Medical Association; Volume: 219; Issue: 8 Linguagem: Inglês

10.2460/javma.2001.219.1089

1943-569X

E. Christopher Orton, Khursheed R. Mama, Peter W. Hellyer, Timothy B. Hackett,

Coronary Artery Anomalies

T etralogy of Fallot is a complex congenital heart defect characterized by right ventricular outflow tract (RVOT) obstruction, ventricular septal defect (VSD), dextropositioned and overriding aorta, and right ventricular hypertrophy.Tetralogy of Fallot is the most important cyanotic heart defect in dogs and accounts for approximately 4% of cardiac malformations in dogs overall. 1 The defect occurs in several breeds of dog, including Keeshond, Poodle, Miniature Schnauzer, Beagle, English Bulldog, Collie, Shetland Sheepdog, and several terrier breeds. 1,2he pathophysiologic consequences of tetralogy depend on the magnitude of RVOT obstruction.With severe RVOT obstruction, the defect allows right-toleft shunt of blood from the right ventricle through the VSD into the systemic circulation.The results are hypoxemia, cyanosis, progressive polycythemia, debilitating weakness and exercise intolerance, and shortened life span.Surgical palliation of tetralogy of Fallot by creation of a systemic-to-pulmonary shunt has been described in dogs. 2,3Long-term palliation of tetralogy of Fallot by these shunts has not been demonstrated in dogs.Definitive open surgical repair is considered the only viable long-term treatment for this defect in human children. 4This report describes the technique for definitive open surgical repair of tetralogy of Fallot in dogs.The intermediate-term outcome of 2 severely affected dogs that underwent this correction is reported.