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CLINICAL OBSERVATIONS ON CRANIOTABES AND RICKETS

1925; American Medical Association; Volume: 29; Issue: 5 Linguagem: Inglês

10.1001/archpedi.1925.04120290024004

1538-3628

Sally Wilson,

Connective tissue disorders research

All through the extensive studies of recent years in rickets, little has been done to investigate craniotabes. In the past, some investigators, notably Kassowitz 1 and later Wieland, 2 concerned themselves with the etiology and pathology of this clinical condition. They found it frequently associated with rickets, and the former believed that craniotabes was a manifestation of rickets and that all rickets, therefore, was congenital in origin. Wieland, 3 Schloss 4 and Finkelstein 5 believed that the early congenital softenings were physiologic in nature and soon disappeared, while new softenings appearing on the hard bone later were "rachitic." Czerny 6 states that craniotabes makes a positive diagnosis of rickets. Marfan 7 says that true craniotabes is of rachitic origin, but believes syphilis to be the most common cause of craniotabes. De Stefano 8 states that craniotabes is a manifestation either of rickets or of inherited syphilis. Thus, we see that