A case of bullous pemphigoid associated with psoriasis vulgaris showing Hailey–Hailey disease‐like histopathological changes in regenerated epidermis without genomic mutation in ATP2C1 or ATP2A2 gene
2014; Wiley; Volume: 29; Issue: 8 Linguagem: Inglês
10.1111/jdv.12521
ISSN1468-3083
AutoresYutaka Iino, Toshiyuki Kano, Fukashi Adachi, Midori Suzuki, Ryuhei Nishikawa, Norito Ishii, Chika Ohata, Minao Furumura, Takahiro Hamada, Takashi Hashimoto,
Tópico(s)Autoimmune Bullous Skin Diseases
ResumoJournal of the European Academy of Dermatology and VenereologyVolume 29, Issue 8 p. 1646-1648 Letter to the Editor A case of bullous pemphigoid associated with psoriasis vulgaris showing Hailey–Hailey disease-like histopathological changes in regenerated epidermis without genomic mutation in ATP2C1 or ATP2A2 gene Y. Iino, Y. Iino Department of Dermatology, Ibaraki Prefectural Central Hospital, Kasama, Ibaraki, JapanSearch for more papers by this authorT. Kano, T. Kano Department of Dermatology, Ibaraki Prefectural Central Hospital, Kasama, Ibaraki, JapanSearch for more papers by this authorF. Adachi, F. Adachi Department of Dermatology, Ibaraki Prefectural Central Hospital, Kasama, Ibaraki, JapanSearch for more papers by this authorM. Suzuki, M. Suzuki Department of Dermatology, Ibaraki Prefectural Central Hospital, Kasama, Ibaraki, JapanSearch for more papers by this authorR. Nishikawa, R. Nishikawa Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorN. Ishii, N. Ishii Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorC. Ohata, C. Ohata Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorM. Furumura, M. Furumura Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorT. Hamada, T. Hamada Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorT. Hashimoto, Corresponding Author T. Hashimoto Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanCorrespondence: T. Hashimoto. E-mail: [email protected]Search for more papers by this author Y. Iino, Y. Iino Department of Dermatology, Ibaraki Prefectural Central Hospital, Kasama, Ibaraki, JapanSearch for more papers by this authorT. Kano, T. Kano Department of Dermatology, Ibaraki Prefectural Central Hospital, Kasama, Ibaraki, JapanSearch for more papers by this authorF. Adachi, F. Adachi Department of Dermatology, Ibaraki Prefectural Central Hospital, Kasama, Ibaraki, JapanSearch for more papers by this authorM. Suzuki, M. Suzuki Department of Dermatology, Ibaraki Prefectural Central Hospital, Kasama, Ibaraki, JapanSearch for more papers by this authorR. Nishikawa, R. Nishikawa Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorN. Ishii, N. Ishii Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorC. Ohata, C. Ohata Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorM. Furumura, M. Furumura Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorT. Hamada, T. Hamada Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanSearch for more papers by this authorT. Hashimoto, Corresponding Author T. Hashimoto Department of Dermatology, Kurume University School of Medicine, and Kurume University Institute of Cutaneous Cell Biology, Kurume, Fukuoka, JapanCorrespondence: T. Hashimoto. E-mail: [email protected]Search for more papers by this author First published: 21 April 2014 https://doi.org/10.1111/jdv.12521Citations: 5Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL No abstract is available for this article. References 1Ohata C, Koga H, Teye K et al. Concurrence of bullous pemphigoid and herpetiform pemphigus with IgG antibodies to desmogleins 1/3 and desmocollins 1-3. Br J Dermatol 2013; 168: 879– 881. 2Mehregan DA, Umbert IJ, Peters MS. Histologic findings of Hailey-Hailey disease in a patient with bullous pemphigoid. 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Envoplakin and periplakin are components of the paraneoplastic pemphigus antigen complex. J Invest Dermatol 1998; 111: 1236– 1238. 9Endo Y, Tsujioka K, Tanioka M et al. Bullous dermatosis associated with IgG antibodies specific for desmocollins. Eur J Dermatol 2010; 20: 620– 625. 10Koerber WA Jr, Price NM, Watson W. Coexistent psoriasis and bullous pemphigoid: a report of six cases. Arch Dermatol 1978; 114: 1643– 1646. Citing Literature Volume29, Issue8August 2015Pages 1646-1648 ReferencesRelatedInformation
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