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BIBTEX RIS

Hyperkalemia alters endothelium-dependent relaxation through non—nitric oxide and noncyclooxygenase pathway: A mechanism for coronary dysfunction due to cardioplegia

1996; Elsevier BV; Volume: 61; Issue: 5 Linguagem: Inglês

10.1016/0003-4975(96)00086-0

ISSN

1552-6259

Autores

Guo‐Wei He, Cheng‐Qin Yang,

Tópico(s)

Heart Failure Treatment and Management

Resumo

Background.Reported results of hyperkalemia (cardioplegia or organ preservation solutions) on endothelial function are contradictory.The endothelium-dependent relaxation is related to three major mechanisms: cyclooxygenase, nitric oxide, and endothelium-derived hyperpolarizing factor (K ÷ channel related).The present study was designed to test the hypothesis that hyperkalemia may alter endothelial function through non-nitric oxide and noncyclooxygenase pathways.

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