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Anion Gap Acidosis Associated with Acetaminophen

2000; American College of Physicians; Volume: 133; Issue: 9 Linguagem: Inglês

10.7326/0003-4819-133-9-200011070-00036

ISSN

1539-3704

Autores

Steven H. Yale,

Tópico(s)

Diet and metabolism studies

Resumo

Letters7 November 2000Anion Gap Acidosis Associated with AcetaminophenSteven H. Yale, MD and Joseph J. Mazza, MDSteven H. Yale, MDMarshfield Clinic; Marshfield, WI 54449 (Yale, Mazza)Search for more papers by this author and Joseph J. Mazza, MDMarshfield Clinic; Marshfield, WI 54449 (Yale, Mazza)Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-133-9-200011070-00036 SectionsAboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail TO THE EDITOR:Pyroglutamic aciduria (5-oxoprolinuria) is caused by a deficiency of gluthathione synthetase of 5-oxoprolinase. This disorder has been reported after the ingestion of therapeutic doses of acetaminophen and has been fatal (1). We report pyroglutamic aciduria developing after acetaminophen–oxycodone treatment.A previously healthy 44-year-old woman presented with a 1-month history of back pain treated with acetaminophen–oxycodone, one to two tablets every 6 hours. Evaluation revealed a muscle strain and a fruity smell on her breath. Notable laboratory values were a CO2 level of 15 mEq/L, anion gap of 25 mEq/L, β-hydroxybutyrate level of 5.3 mEq/L, lactate level of ...References1. Pitt JJ, Hauser S. Transient 5-oxoprolinuria and high anion gap metabolic acidosis: clinical and biochemical findings in eleven subjects. Clin Chem. 1998;44:1497-503. CrossrefMedlineGoogle Scholar2. Spielberg SP. In vitro assessment of pharmacogenetic susceptibility to toxic drug metabolites in humans. Fed Proc. 1984;43:2308-13. MedlineGoogle Scholar3. Spielberg SP. Acetaminophen toxicity in lymphocytes heterozygous for glutathione synthetase deficiency. Can J Physiol Pharmacol. 1985;63:468-71. CrossrefMedlineGoogle Scholar4. Creer MH, Lau BW, Jones JD, Chan KM. Pyroglutamic acidemia in an adult patient. Clin Chem. 1989;35:684-6. CrossrefMedlineGoogle Scholar5. Pitt JJ, Brown GK, Clift V, Christodoulou J. Atypical pyroglutamic aciduria: possible role of paracetamol. J Inherit Metab Dis. 1990;13:755-6. CrossrefMedlineGoogle Scholar Author, Article, and Disclosure InformationAuthors: Steven H. Yale, MD; Joseph J. Mazza, MDAffiliations: Marshfield Clinic; Marshfield, WI 54449 (Yale, Mazza) PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byIatrogenic and Poison-Derived Acid Base DisordersAcidose métabolique à trou anionique élevé dans un contexte d’intoxication chronique au paracétamol (acétaminophène)Translational biomarkers of acetaminophen-induced acute liver injuryWhat is the clinical significance of 5-oxoproline (pyroglutamic acid) in high anion gap metabolic acidosis following paracetamol (acetaminophen) exposure?An Unusual Cause of High Anion Gap Metabolic AcidosisAcetaminophenSevere Anion Gap Metabolic Acidosis From Acetaminophen Use Secondary to 5-Oxoproline (Pyroglutamic Acid) AccumulationRecurrent anion gap metabolic acidosis in a woman with vertebral disc diseaseFour nephrology myths debunkedUnderstanding lactic acidosis in paracetamol (acetaminophen) poisoningAcid-Base Disorders Secondary to PoisoningParacetamol-induced 5-oxoprolinuira and high anion gap metabolic acidosis in a child on a ketogenic dietProfound metabolic acidosis and oxoprolinuria in an adultRecurrent High Anion Gap Metabolic Acidosis Secondary to 5-Oxoproline (Pyroglutamic Acid)Acetaminophen-Induced Anion Gap Metabolic Acidosis and 5-Oxoprolinuria (Pyroglutamic Aciduria) Acquired in Hospital 7 November 2000Volume 133, Issue 9Page: 752-753KeywordsAnionsCell metabolismGlutathioneIngestionLigasesMusclesResearch laboratoriesSalicylatesSmellUrine ePublished: 7 November 2000 Issue Published: 7 November 2000 Copyright & PermissionsCopyright © 2000 by American College of Physicians. All Rights Reserved.PDF downloadLoading ...

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