Artigo Revisado por pares

Very late onset Friedreich's ataxia without cardiomyopathy is associated with limited GAA expansion in the X25 gene

1997; Lippincott Williams & Wilkins; Volume: 49; Issue: 4 Linguagem: Inglês

10.1212/wnl.49.4.1153

ISSN

1526-632X

Autores

Cinzia Gellera, Davide Pareyson, Barbara Castellotti, Franca Mazzucchelli, B. Zappacosta, Massimo Pandolfo, Stefano Di Donato,

Tópico(s)

Mitochondrial Function and Pathology

Resumo

Molecular analysis of spinocerebellar ataxias revealed a pathologic GAA expansion in the gene encoding frataxin in six adult patients from three families. These patients, carrying expanded alleles in the low-range size, had an exceptionally late onset and lacked cardiomyopathy, pointing to phenotypic variability of Friedreich's ataxia. Both mitotic and gametic instability of the expanded triplet repeat were present in these families.

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