Artigo Revisado por pares

The Provant® Wound Closure System Induces Activation of p44/42 MAP Kinase in Normal Cultured Human Fibroblasts

2002; Wiley; Volume: 961; Issue: 1 Linguagem: Inglês

10.1111/j.1749-6632.2002.tb03076.x

ISSN

1749-6632

Autores

Terri L. Gilbert, Nicole Griffin, John R. Moffett, Mary C. Ritz, Frank R. George,

Tópico(s)

Cellular Mechanics and Interactions

Resumo

Annals of the New York Academy of SciencesVolume 961, Issue 1 p. 168-171 The Provant® Wound Closure System Induces Activation of p44/42 MAP Kinase in Normal Cultured Human Fibroblasts TERRI L. GILBERT, TERRI L. GILBERT Regenesis Biomedical Inc., Scottsdale, Arizona 85257, USASearch for more papers by this authorNICOLE GRIFFIN, NICOLE GRIFFIN Department of Microbiology, Arizona State University, Tempe, Arizona, USASearch for more papers by this authorJOHN MOFFETT, JOHN MOFFETT Harrington Arthritis Research Center, Phoenix, Arizona 85006, USASearch for more papers by this authorMARY C. RITZ, MARY C. RITZ Regenesis Biomedical Inc., Scottsdale, Arizona 85257, USASearch for more papers by this authorFRANK R. GEORGE, Corresponding Author FRANK R. GEORGE Regenesis Biomedical Inc., Scottsdale, Arizona 85257, USAAddress for correspondence: Frank R. George, Regenesis Biomedical Inc., 1435 N. Hayden Road, Scottsdale, AZ 85257. Voice: 480-970-4970; fax: 480-970-8792; [email protected].Search for more papers by this author TERRI L. GILBERT, TERRI L. GILBERT Regenesis Biomedical Inc., Scottsdale, Arizona 85257, USASearch for more papers by this authorNICOLE GRIFFIN, NICOLE GRIFFIN Department of Microbiology, Arizona State University, Tempe, Arizona, USASearch for more papers by this authorJOHN MOFFETT, JOHN MOFFETT Harrington Arthritis Research Center, Phoenix, Arizona 85006, USASearch for more papers by this authorMARY C. RITZ, MARY C. RITZ Regenesis Biomedical Inc., Scottsdale, Arizona 85257, USASearch for more papers by this authorFRANK R. GEORGE, Corresponding Author FRANK R. GEORGE Regenesis Biomedical Inc., Scottsdale, Arizona 85257, USAAddress for correspondence: Frank R. George, Regenesis Biomedical Inc., 1435 N. Hayden Road, Scottsdale, AZ 85257. Voice: 480-970-4970; fax: 480-970-8792; [email protected].Search for more papers by this author First published: 24 January 2006 https://doi.org/10.1111/j.1749-6632.2002.tb03076.xCitations: 17Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onEmailFacebookTwitterLinkedInRedditWechat REFERENCES 1 Schaffer, C.J. & L.B. Nanney. 1996. Cell biology of wound healing. Int. Rev. Cytol. 169: 151–181. 10.1016/S0074-7696(08)61986-5 CASPubMedWeb of Science®Google Scholar 2 George, F.R., R. Gallegos, W.D. Dachman, et al. 2002. Clinical efficacy of cell proliferation induction (CPI): a novel biophysical stimulus for accelerated wound Healing to closure. Wounds. In press. Google Scholar 3 George, F.R., R.J. Lukas, J. Moffett, et al. 2002. In vitro mechanisms of cell proliferation induction (cpi): a novel bioactive treatment for accelerating wound healing. Wounds 14: 107–115. Web of Science®Google Scholar 4 Cleary, S.F., L.M. Liu & R.E. Merchant. 1990. In vitro lymphocyte proliferation induced by radio-frequency electromagnetic radiation under isothermal conditions. Bioelectromagnetics 11: 47–56. 10.1002/bem.2250110107 CASPubMedWeb of Science®Google Scholar 5 Cleary, S.F., L.M. Liu & R.E. Merchant. 1990. Glioma proliferation modulated in vitro by isothermal radiofrequency radiation exposure. Radiat. Res. 121: 38–45. 10.2307/3577561 CASPubMedWeb of Science®Google Scholar 6 Dent, P., W.D. Jarvis, M.J. Birrer, et al. 1998. The roles of signaling by the p42/p44 mitogen-activated protein (MAP) kinase pathway: a potential route to radio- and chemo-sensitization of tumor cells resulting in the induction of apoptosis and loss of clonogenicity. Leukemia 12: 1843–1850. 10.1038/sj.leu.2401222 CASPubMedWeb of Science®Google Scholar 7 Pelech, S.L. & D.L. Charest. 1995. MAP kinase-dependent pathways in cell cycle control. Prog. Cell Cycle Res. 1: 33–52. 10.1007/978-1-4615-1809-9_4 CASPubMedGoogle Scholar 8 Davis, R.J. 1995. Transcriptional regulation by MAP kinases. Mol. Reprod. Dev. 42: 459–467. 10.1002/mrd.1080420414 CASPubMedWeb of Science®Google Scholar 9 Cobb, M. H. 1999. MAP kinase pathways. Prog. Biophys. Mol. Biol. 71: 479–500. 10.1016/S0079-6107(98)00056-X CASPubMedWeb of Science®Google Scholar Citing Literature Volume961, Issue1REPARATIVE MEDICINE: GROWING TISSUES AND ORGANSJune 2002Pages 168-171 ReferencesRelatedInformation

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