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The Molecular Basis of Anticholinesterase Actions on Nicotinic and Glutamatergic Synapses a

1987; Wiley; Volume: 505; Issue: 1 Linguagem: Inglês

10.1111/j.1749-6632.1987.tb51294.x

1749-6632

Yasco Aracava, S.S. Deshpande, D L Rickett, A. BROSSI, Bernhard Schönenberger, Edson X. Albuquerque,

Ion channel regulation and function

Annals of the New York Academy of SciencesVolume 505, Issue 1 p. 226-255 The Molecular Basis of Anticholinesterase Actions on Nicotinic and Glutamatergic Synapsesa Y. ARACAVA, Y. ARACAVA Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201Search for more papers by this authorS. S. DESHPANDE, S. S. DESHPANDE Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201Search for more papers by this authorD. L. RICKETT, Corresponding Author D. L. RICKETT Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201 Neurotoxicology Branch, United States Army Research Institute of Chemical Defense, Aberdeen Proving Ground, Aberdeen, Maryland 21010. Laboratory of Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, Maryland 20892. To whom reprint requests should be sent.Search for more papers by this authorA. BROSSI, Corresponding Author A. BROSSI Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201 Neurotoxicology Branch, United States Army Research Institute of Chemical Defense, Aberdeen Proving Ground, Aberdeen, Maryland 21010. Laboratory of Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, Maryland 20892. To whom reprint requests should be sent.Search for more papers by this authorB. SCHÖNENBERGER, Corresponding Author B. SCHÖNENBERGER Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201 Neurotoxicology Branch, United States Army Research Institute of Chemical Defense, Aberdeen Proving Ground, Aberdeen, Maryland 21010. Laboratory of Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, Maryland 20892. To whom reprint requests should be sent.Search for more papers by this authorE. X. ALBUQUERQUE, Corresponding Author E. X. ALBUQUERQUE Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201 Neurotoxicology Branch, United States Army Research Institute of Chemical Defense, Aberdeen Proving Ground, Aberdeen, Maryland 21010. Laboratory of Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, Maryland 20892. To whom reprint requests should be sent.Search for more papers by this author Y. ARACAVA, Y. ARACAVA Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201Search for more papers by this authorS. S. DESHPANDE, S. S. DESHPANDE Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201Search for more papers by this authorD. L. RICKETT, Corresponding Author D. L. RICKETT Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201 Neurotoxicology Branch, United States Army Research Institute of Chemical Defense, Aberdeen Proving Ground, Aberdeen, Maryland 21010. Laboratory of Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, Maryland 20892. To whom reprint requests should be sent.Search for more papers by this authorA. BROSSI, Corresponding Author A. BROSSI Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201 Neurotoxicology Branch, United States Army Research Institute of Chemical Defense, Aberdeen Proving Ground, Aberdeen, Maryland 21010. Laboratory of Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, Maryland 20892. To whom reprint requests should be sent.Search for more papers by this authorB. SCHÖNENBERGER, Corresponding Author B. SCHÖNENBERGER Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201 Neurotoxicology Branch, United States Army Research Institute of Chemical Defense, Aberdeen Proving Ground, Aberdeen, Maryland 21010. Laboratory of Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, Maryland 20892. To whom reprint requests should be sent.Search for more papers by this authorE. X. ALBUQUERQUE, Corresponding Author E. X. ALBUQUERQUE Department of Pharmacology and Experimental Therapeutics University of Maryland School of Medicine Baltimore, Maryland 21201 Neurotoxicology Branch, United States Army Research Institute of Chemical Defense, Aberdeen Proving Ground, Aberdeen, Maryland 21010. Laboratory of Chemistry, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, Maryland 20892. To whom reprint requests should be sent.Search for more papers by this author First published: August 1987 https://doi.org/10.1111/j.1749-6632.1987.tb51294.xCitations: 19 a This research was supported by United States Army Medical Research and Development Command Contract DAMD17–84-C-4219 and by United States Army Research Office Grant DAAG 29–85-K-0090. 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Ther. 239: 279– 285. 62 Idriss, M. & E. X. Albuquerque. 1985. Phencyclidine (PCP) blocks glutamate-activated postsynaptic currents. FEBS Lett. 189: 150– 156. Citing Literature Volume505, Issue1Myasthenia Gravis: Biology and TreatmentAugust 1987Pages 226-255 ReferencesRelatedInformation