Artigo Revisado por pares

Short term fingolimod treatment decreases soluble VLA 4 levels in MS patients

2014; Elsevier BV; Volume: 275; Issue: 1-2 Linguagem: Inglês

10.1016/j.jneuroim.2014.08.610

ISSN

1872-8421

Autores

Erdem Tüzün, Canan Ulusoy, Selin Turan, Arda Örçen, Berrak Yetimler, Deniz Akbaş-Demir, Melike Küçükerden, Hazal Haytural, Recai Türkoğlu,

Tópico(s)

Immune Response and Inflammation

Resumo

Multiple sclerosis (MS) is an immune mediated disorder of the central nervous system. T-cells and other immune effector cells play crucial roles in MS pathogenesis by crossing the brain–blood barrier and consequently destroying the myelin sheat and axons. Fingolimod (FTY720) is an oral sphingosine-1-phosphate (S1P) receptor modulator, approved for treatment of MS. Resting T and B lymphocytes express elevated levels of S1P receptor and lymphocyte migration from the lymphoid organs to the brain depends on the activity of this receptor.

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