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Why do we faint?

2001; Wiley; Volume: 24; Issue: 8 Linguagem: Galês

10.1002/mus.1102

1097-4598

Horacio Kaufmann, R. Hainsworth,

Neurological disorders and treatments

Muscle & NerveVolume 24, Issue 8 p. 981-983 Editorial Why do we faint? Horacio Kaufmann MD, Corresponding Author Horacio Kaufmann MD [email protected] Department of Neurology, Autonomic Nervous System Laboratory, Annenberg 2, Box 1052, Mount Sinai School of Medicine, New York, New York 10029, USADepartment of Neurology, Autonomic Nervous System Laboratory, Annenberg 2, Box 1052, Mount Sinai School of Medicine, New York, New York 10029, USASearch for more papers by this authorRoger Hainsworth MB, PhD, DSc, Roger Hainsworth MB, PhD, DSc Institute for Cardiovascular Research, University of Leeds, Leeds, UKSearch for more papers by this author Horacio Kaufmann MD, Corresponding Author Horacio Kaufmann MD [email protected] Department of Neurology, Autonomic Nervous System Laboratory, Annenberg 2, Box 1052, Mount Sinai School of Medicine, New York, New York 10029, USADepartment of Neurology, Autonomic Nervous System Laboratory, Annenberg 2, Box 1052, Mount Sinai School of Medicine, New York, New York 10029, USASearch for more papers by this authorRoger Hainsworth MB, PhD, DSc, Roger Hainsworth MB, PhD, DSc Institute for Cardiovascular Research, University of Leeds, Leeds, UKSearch for more papers by this author First published: 29 June 2001 https://doi.org/10.1002/mus.1102Citations: 28Read the full textAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onEmailFacebookTwitterLinkedInRedditWechat REFERENCES 1Barcroft H, Edholm O. On the vasodilatation in human skeletal muscle during post-hemorrhagic fainting. J Physiol 1945; 104: 161–175. 2Blair D, Glover W, Greenfield A, Roddie I. Excitation of cholinergic vasodilator nerves to human skeletal muscles during emotional stress. J Physiol 1959; 148: 633–647. 3Dietz NM, Rivera JM, Eggener SE, Fix RT, Warner DO, Joyner MJ. Nitric oxide contributes to the rise in forearm blood flow during mental stress in humans. J Physiol (Lond) 1994; 480: 361–368. 4Ekman P, Levenson RW, Friesen WV. Autonomic nervous system activity distinguishes among emotions. Science 1983; 221: 1208–1210. 5El-Bedawi KM, Wahbha MA, Hainsworth R. Cardiac pacing does not improve orthostatic tolerance in patients with vasovagal syncope. Clin Auton Res 1994; 4: 233–237. 6El-Sayed H, Hainsworth R. Salt supplement increases plasma volume and orthostatic tolerance in patients with unexplained syncope. Heart 1996; 75: 134–140. 7Fitzpatrick AP, Banner N, Cheng A, Yacoub M, Sutton R. Vasovagal reactions may occur after orthotopic heart transplantation. J Am Coll Cardiol 1993; 21: 1132–1137. 8Glover W, Greenfield A, Shanks R. The contribution made by adrenaline to the vasodilation in the human forearm during emotional stress. J Physiol 1962; 161: 42P–43P. 9Goldstein D. Stress response patterns. In: Stress, catecholamines, and cardiovascular disease. New York: Oxford University Press; 1995. p 287–328. 10Hainsworth R, El-Bedawi KM. Orthostatic tolerance in patients with unexplained syncope. Clin Auton Res 1994; 4: 239–244. 11Kaufmann H, Berman J, Oribe E, Oliver J. Possible increase in the synthesis of endothelial derived relaxing factor (EDRF) during vasovagal syncope. Clin Auton Res 1993; 3: 69. 12Kaufmann H, Oribe E, Oliver JA. Plasma endothelin during upright tilt: relevance for orthostatic hypotension? Lancet 1991; 338: 1542–1545. 13Koizumi K, Kollai M. Control of reciprocal and non-reciprocal action of vagal and sympathetic efferents: study of centrally induced reactions. J Auton Nerv Syst 1981; 3: 483–501. 14Lewis T. Lecture on vasovagal syncope and the carotid sinus mechanism. BMJ 1932; 1: 873–876. 15Mtinangi BL, Hainsworth R. Increased orthostatic tolerance following moderate exercise training in patients with unexplained syncope. Heart 1998; 80: 596–600. 16Oberg B, Thoren P. Increased activity in left ventricular receptors during hemorrhage or occlusion of caval veins in the cat. A possible cause of the vaso-vagal reaction. Acta Physiol Scand 1972; 85: 164–173. 17Uvnas B. Cholinergic vasodilator nerves. Fed Proc 1966; 25: 1618–1622. 18Wallin BG, Sundlof G. Sympathetic outflow to muscles during vasovagal syncope. J Auton Nerv Syst 1982; 6: 287–291. Citing Literature Volume24, Issue8August 2001Pages 981-983 ReferencesRelatedInformation