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Síndrome de baboon

2004; Elsevier BV; Volume: 32; Issue: 1 Linguagem: Inglês

10.1016/s0301-0546(04)79223-x

1578-1267

L Sánchez-Morillas, Marco Martos, M. Rodríguez Mosquera, C. Iglesias Cadarso, Luis González Sánchez, A.R. Domínguez Lázaro,

Venomous Animal Envenomation and Studies

Andersen et al described baboon syndrome in 1984. It was characterized by a clinical presentation of systemic contact dermatitis with pruritic and confluent maculopapular light-red eruption, localized in the gluteal area and the major flexures, developed several hours or days after drug or agent contact. This syndrome has a pathognomonic distribution but its cause has not been elucidated yet. Histopathology of the lesions shows non-specific features of dermatitis. Several drugs have been previously described as responsible for the Baboon syndrome origin. Mercury is the most frequent implicated agent; other agents are nickel, different antibiotics, heparine, aminophylline, pseudoephedrine, terbinafine and immunoglobulins