Artigo Acesso aberto Revisado por pares

Effect of Eculizumab on Hemolysis and Transfusion Requirements in Patients with Paroxysmal Nocturnal Hemoglobinuria

2004; Massachusetts Medical Society; Volume: 350; Issue: 6 Linguagem: Inglês

10.1056/nejmoa031688

ISSN

1533-4406

Autores

Peter Hillmen, Claire Hall, Judith Marsh, Modupe Elebute, Michael P. Bombara, Beth E. Petro, Matthew Cullen, Stephen J. Richards, Scott A. Rollins, Christopher F. Mojcik, Russell P. Rother,

Tópico(s)

Blood groups and transfusion

Resumo

Paroxysmal nocturnal hemoglobinuria (PNH) arises from a somatic mutation of the PIG-A gene in a hematopoietic stem cell and the subsequent production of blood cells with a deficiency of surface proteins that protect the cells against attack by the complement system. We tested the clinical efficacy of eculizumab, a humanized antibody that inhibits the activation of terminal complement components, in patients with PNH.

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