Artigo Acesso aberto Revisado por pares

A potent antagonist antibody targeting connexin hemichannels alleviates Clouston syndrome symptoms in mutant mice

2020; Elsevier BV; Volume: 57; Linguagem: Inglês

10.1016/j.ebiom.2020.102825

ISSN

2352-3964

Autores

Yuanyuan Kuang, Veronica Zorzi, Damiano Buratto, Gaia Ziraldo, Flavia Mazzarda, Chiara Peres, Chiara Nardin, Anna Maria Salvatore, Francesco Chiani, Ferdinando Scavizzi, Marcello Raspa, Min Qiang, Youjun Chu, Xiaojie Shi, Yu Li, Lili Liu, Yaru Shi, Francesco Zonta, Guang Yang, Richard A. Lerner, Fabio Mammano,

Tópico(s)

Erythrocyte Function and Pathophysiology

Resumo

Numerous currently incurable human diseases have been causally linked to mutations in connexin (Cx) genes. In several instances, pathological mutations generate abnormally active Cx hemichannels, referred to also as "leaky" hemichannels. The goal of this study was to assay the in vivo efficacy of a potent antagonist antibody targeting Cx hemichannels.

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