Heightened resistance to host type 1 interferons characterizes HIV-1 at transmission and after antiretroviral therapy interruption
2021; American Association for the Advancement of Science; Volume: 13; Issue: 576 Linguagem: Inglês
10.1126/scitranslmed.abd8179
ISSN1946-6242
AutoresMarcos V. P. Gondim, Scott Sherrill-Mix, Frédéric Bibollet‐Ruche, Ronnie M. Russell, Stephanie Trimboli, Andrew G. Smith, Yingying Li, Weimin Liu, Alexa N. Avitto, Julia C. DeVoto, Andrew Connell, Angharad E. Fenton-May, Pierre Pellegrino, Ian Williams, Emmanouil Papasavvas, Julio C. C. Lorenzi, D. Brenda Salantes, Felicity Mampe, M. Alexandra Monroy, Yehuda Z. Cohen, Sonya L. Heath, Michael S. Saag, Luis J. Montaner, Ronald G. Collman, Janet D. Siliciano, Robert F. Siliciano, Lindsey J. Plenderleith, Paul M. Sharp, Marina Caskey, Michel C. Nussenzweig, George M. Shaw, Persephone Borrow, Katharine J. Bar, Beatrice H. Hahn,
Tópico(s)Immune Cell Function and Interaction
ResumoInterferon-mediated pressures vary throughout HIV-1 infection but are the highest during viral rebound after treatment interruption.
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