Autoantigen-specific CD4+ T cells acquire an exhausted phenotype and persist in human antigen-specific autoimmune diseases
2024; Cell Press; Volume: 57; Issue: 10 Linguagem: Inglês
10.1016/j.immuni.2024.08.005
ISSN1097-4180
AutoresCarina Saggau, Petra Bächer, Daniela Esser, Mahdi Rasa, Silja Meise, Nicola Mohr, Nora Kohlstedt, Andreas Hutloff, Sarah-Sophie Schacht, Justina Dargvainiene, Gabriela Rios Martini, Klarissa Hanja Stürner, Ina Schröder, Robert Markewitz, Johannes Hartl, Maria Hastermann, Ankelien Duchow, Patrick Schindler, Mareike Becker, Carolin Bautista, Judith Gottfreund, Jörn Walter, Julia K. Polansky, Mingxing Yang, Reza Naghavian, Mareike Wendorff, Ev-Marie Schuster, Andreas Dahl, A. Petzold, Susanne Reinhardt, André Franke, Marek Wieczorek, Lea Henschel, Daniel Berger, Guido Heine, Maike M. Holtsche, Vivien Häußler, Christian Peters, Enno Schmidt, Simon Fillatreau, Dirk H. Busch, Klaus‐Peter Wandinger, Kilian Schober, Roland Martinꝉ, Friedemann Paul, Frank Leypoldt, Alexander Scheffold,
Tópico(s)Atherosclerosis and Cardiovascular Diseases
ResumoPro-inflammatory autoantigen-specific CD4
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