Genome-wide Association Study Identifies Genetic Variation in Neurocan as a Susceptibility Factor for Bipolar Disorder
2011; Elsevier BV; Volume: 88; Issue: 3 Linguagem: Inglês
10.1016/j.ajhg.2011.03.001
ISSN1537-6605
AutoresSven Cichon, Thomas W. Mühleisen, Franziska Degenhardt, Manuel Mattheisen, Xavier Miró, Jana Strohmaier, Michael Steffens, Christian Meesters, Stefan Herms, Moritz Weingarten, Lutz Priebe, Britta Haenisch, Michael P. Alexander, Jennifer Vollmer, René Breuer, Christine Schmäl, Peter Teßmann, Susanne Moebus, H.-Erich Wichmann, Stefan Schreiber, Bertram Müller‐Myhsok, Susanne Lucae, Stéphane Jamain, Marion Leboyer, Frank Bellivier, Bruno Étain, Chantal Henry, Jean‐Pierre Kahn, Simon Heath, Marian Hamshere, Michael O’Donovan, Michael J. Owen, Nick Craddock, Markus Schwarz, Helmut Vedder, Jutta Kammerer-Ciernioch, Andreas Reif, Johanna Sasse, Michael Bauer, Martin Hautzinger, A. Jordan Wright, Philip B. Mitchell, Peter R. Schofield, Grant W. Montgomery, Sarah E. Medland, Scott D. Gordon, Nicholas G. Martin, Ómar Gústafsson, Ole A. Andreassen, Srdjan Djurovic, Engilbert Sigurðsson, Stacy Steinberg, Hreinn Stefánsson, Kāri Stefánsson, Lejla Pojskić, L Oruc, Fabio Rivas, Fermín Mayoral, А. Г. Чучалин, Gulja Babadjanova, Tiganov As, Galina Pantelejeva, Л. И. Абрамова, Maria Grigoroiu‐Serbânescu, Carmen C. Diaconu, Piotr M. Czerski, Joanna Hauser, Andreas Zimmer, Mark Lathrop, Thomas G. Schulze, Thomas F. Wienker, Johannes Schumacher, Wolfgang Maier, Peter Propping, Marcella Rietschel, Markus M. Nöthen,
Tópico(s)Genetic Associations and Epidemiology
Resumo(The American Journal of Human Genetics 88, 372–381; March 11, 2011) In the original version of this paper, affiliation 44 was incorrectly attributed to Dr. Cichon and Dr. Muhleisen. The correct affiliation for both authors is 45, indicating co-authorship. This has been corrected both in print and online, and the correct author list appears here as well. Genome-wide Association Study Identifies Genetic Variation in Neurocan as a Susceptibility Factor for Bipolar DisorderCichon et al.The American Journal of Human GeneticsFebruary 24, 2011In BriefWe conducted a genome-wide association study (GWAS) and a follow-up study of bipolar disorder (BD), a common neuropsychiatric disorder. In the GWAS, we investigated 499,494 autosomal and 12,484 X-chromosomal SNPs in 682 patients with BD and in 1300 controls. In the first follow-up step, we tested the most significant 48 SNPs in 1729 patients with BD and in 2313 controls. Eight SNPs showed nominally significant association with BD and were introduced to a meta-analysis of the GWAS and the first follow-up samples. Full-Text PDF Open Archive
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